{"id":10649,"date":"2020-10-01T17:36:24","date_gmt":"2020-10-01T16:36:24","guid":{"rendered":"https:\/\/nutrishield.com\/?p=10649"},"modified":"2021-06-24T11:58:53","modified_gmt":"2021-06-24T10:58:53","slug":"covid-2-age-risk","status":"publish","type":"post","link":"https:\/\/staging.nutrishield.com\/?p=10649","title":{"rendered":"Covid Review 2 Age Risk"},"content":{"rendered":"
\n
\n\t

Countering COVID \u2013 A 5-part Review by Colin Rose October 2020 Part 2 Age Risk<\/h1>\n

Colin Rose, the author of this series of articles, is a Senior Associate of the Royal Society of Medicine (RSM). He draws on recent updates from multiple sources including both the US and European Centres for Disease Prevention and Control. He researches and writes for Uni-Vite Healthcare Ltd.<\/p>\n

In this new five-part series, the emphasis is both on how to improve your chances of a mild outcome and evidence-based ideas for prevention.<\/p>\n<\/div>\n<\/div><\/div>

\n
\n\t

Countering COVID Part 2 \u2013 Age is not the top risk factor<\/h1>\n

Accumulated damage is the risk, not simply age in years<\/h3>\n

Age has been cited as the top risk factor for COVID-19. On paper that is true \u2013 people over 60 are many times more likely to have a bad outcome.<\/p>\n

But that\u2019s not because of the passing of the years. It\u2019s because, as we get older, we accumulate damage. Damage to DNA, damage to the immune system, damage to mitochondria (the tiny \u2018power plants\u2019 in almost every cell). This damage increases vulnerability to both illness and the severity of that illness.<\/p>\n

Repair mechanisms need to be boosted<\/h3>\n

Fortunately, the body has repair mechanisms to counteract that damage and they normally work well when you are younger. But those repair mechanisms generally begin to weaken as we get older \u2013 inflammation increases and what we call \u2018age related illnesses\u2019 begin to surface.<\/p>\n

But they are not age related, they are damage related.<\/p>\n

What if we could boost those repair mechanisms back up to a younger state? Then inflammation would be damped down, the onset of illness would be postponed, or even halted, and we would \u2013 literally \u2013 age slower.<\/p>\n

Recent research shows we can delay ageing<\/h3>\n

We now know how to do that \u2013 thanks to researchers into ageing at leading universities and medical centres, including Oxford, Cambridge, University College London and Harvard.<\/p>\n

They have recently made important breakthroughs in understanding the link between inflammation, ageing and disease \u2013 and how to interrupt those links. I believe this research has very important implications for avoiding a bad coronavirus outcome.<\/p>\n

So, I hope you will forgive me mentioning my new book Delay Ageing<\/em> which describes these discoveries. It follows a 2019 article in the leading science journal Nature stating:<\/p>\n

\n

\u201cWe are now entering an exciting era for research on ageing. This era holds unprecedented promise for increasing human health-span: preventing, delaying or\u2014in some cases\u2014reversing many of the causes of ageing based on new scientific discoveries.\u201d<\/p>\n<\/blockquote>\n

Read the introduction to Colin Rose\u2019s new book Delay Ageing<\/h3>\n

You can decide whether Delay Ageing is for you \u2013 and how its recommendations can help in the current pandemic situation \u2013 by clicking this link now to read the Introduction.<\/p>\n

Delay Ageing book<\/a><\/p><\/blockquote>\n